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Biomedical subjects

E Navarro

Publications and source records attributed to E Navarro.

At least 127 records · Page 7Linked to original sources
PubMed1980

Induction of anaesthesia with etomidate: haemodynamic study of 36 patients.

The haemodynamic effects of anaesthetic induction with i.v. etomidate have been assessed in 36 patients. The variables studied were recorded under basal conditions and 3 and 10 min after induction, before surgical stimulus. There was a reduction in cardiac output, stroke volume and arterial pressure, and a compensating increase in heart rate. Pulmonary arterial pressure and vascular resistance, central venous pressure and pulmonary capillary wedge pressure were virtually unchanged. Although etomidate has a negative inotropic effect, the variables which were depressed remained at all times within acceptable limits.

Anesthesia, Intravenous↗
PubMed1979

A betamimetic drug and human fetal lung maturation.

In a double-blind and randomized study the administration of a betamimetic drug to the mother from week 33 to 35 produces a significant increase in amniotic fluid palmitic acid levels. We suggest that the administration of a betamimetic drug during pregnancy to the mother can be useful in producing an acceleration of the fetal lung maturation.

Adult↗
PubMed1977

Orciprenaline and fetal lung maturation in rabbits.

The palmitic and stearic acids and the palmitic/stearic ratio in fetal rabbit lungs were studied after orciprenaline administration to the mother from the 26th to the 31st day of gestation. The results were significantly higher in the treated groups than in the control group for the palmitic acid and palmitic/stearic ratio.

Animals↗
PubMed1976

Interferon induction and sensitivity as correlates to virulence of Venezuelan encephalitis viruses for hamsters.

Venezuelan encephalitis (VEE) virus strains, which differ in virulence for adult hamsters, were compared with respect to (a) sensitivity to hamster interferon (IF) in vitro and in vivo and (b) induction of IF in plasma and target tissues (spleen, bone marrow and brain) following subcutaneous inoculation. In vitro, in cultures of a continuous line of hamster kidney cells, hamster interferon inhibited the replication of a benign VEE strain (BeAr 35,645) more than another benign strain (TC-83 vaccine) or two hamster-virulent strains (68 U 201 and Trinidad donkey). In vivo, in hamsters given poly I: poly C 24 hours before virus to induce interferon formation, BeAr 35,645 and TC-83 virus infections were prevented more frequently than infections with virulent strains Trinidad donkey and 68U201. Benign VEE strains BeAr 35,645 and TC-83 induced only slightly lower concentrations of IF in plasma, bone marrow, spleen and brain than virulent strains Trinidad donkey, 63Z21 and 68U201. However, concentrations of infectious BeAr 35,645 virus were significantly lower in these tissues than virulent strains, resulting in higher ratios of IF: invectious virus, suggesting efficient interferon induction. Benign strain TC-83 showed irregular relationships between IF and infectious virus in plasma or blood and tissues, Splenectomy significantly depressed plasma plasma IF responses to TC-83 virus 20 to 30 hours after inoculation. Interferon appears to be a factor that influences virulence of VEE viruses for hamsters.

Animals↗
PubMed1976

[Abdominal complications of the ventriculo peritoneal derivations].

Thirty-nine ventriculo-peritoneal shunts in children have been analyzed since 1962 to 1976 at the Neurological Institute in Montevideo. In these cases there were 8 important abdominal complications (20%): 7 were associated with ventriculitis, all with infiltration or fistula on the trajet, 3 cases were operated on because of intestinal occlusion, 1 of them with perforative acute peritonitis. In the others, gastric distention, abdominal distention, and transient ileus were found. Five patients died, 4 of them with ventriculitis, 3 children who had suffered ventriculitis, survived. They overcame their hydrocephalus by means of ventriculo-atrial shunts, afterwards their infection was cured. From the result of the test of these cases it is gathered in view of a ventriculo-peritoneal shunt disfunction, that it is necessary to: 1) As far as possible rule out ventriculitis. 2) In cases in which ventriculitis should be proved to exist and with a peritoneal reject, after the infection has been cured, one should not insist with the ventriculo-peritoneal diversion, because in this case the possibility of the relapse taking into account the abdominal intolerance, increases. After the ventriculitis should be got over with a proper bacteriological control, a ventriculo-atrial shunt must be performed. 3) The changing of the form of the divertion system is considered immediately after an abdominal aseptic intolerance is present (ileus, ascites, etc.), due to the double risk of intestinal occlusion or ventriculitis.

Cerebrospinal Fluid Shunts↗