Biomedical subjects

E Colon

Publications and source records attributed to E Colon.

31 records · Page 2Linked to original sources

PubMed1987

Insufficiency of carotid and vertebral flow velocity in cerebrovascular disease: a continuous wave Doppler study.

In patients with cerebrovascular disease, quantitative Continuous Wave Doppler sonograms were made of carotid and vertebral arteries. In transient ischaemic attacks (TIA) all flow velocity insufficiencies were found to be related to stenosis. In stroke, however, a relatively high percentage of insufficiencies proved not to be related to stenosis. A disturbed autoregulation seems the most acceptable explanation.

Aged↗

PubMed1987

Medical complications and medical management of bulimia.

The syndrome of bulimia is a common disorder and can be associated with serious psychological and physical morbidity. Unfortunately, many patients are reluctant to discuss their symptoms with their physicians and few clues can be found on physical examination to aid in the diagnosis. Possible physical signs include ulceration or scarring of the dorsum of the hand, salivary gland hypertrophy, and dental enamel erosion. In laboratory testing it is fairly common for patients with active bulimia to have fluid and electrolyte abnormalities, particularly hypokalemic alkalosis, and some also have elevated serum amylase levels. Rare complications include myopathies from misuse of ipecac, ruptured esophagus and pneumomediastinum associated with vomiting, and subtle abnormalities in neuroendocrine regulatory systems. Medical management including monitoring of fluid and electrolyte balance is essential during treatment.

Bulimia↗

PubMed1985

A neurophysiological study of children with the cerebro-hepato-renal syndrome of Zellweger.

The aim of this study was to describe the EEGs, brainstem auditory evoked potentials and somatosensory evoked potentials obtained in eleven children with the Zellweger syndrome. In six out of the eleven patients BAEPs and in five of them SSEPs were performed. Severe abnormalities could be demonstrated, reflecting diffuse cerebral dysfunction as well as long myelinated fiber tracts dysfunction. In four of six patients no response was obtained by BAEPs and in two the central conduction time was delayed. The specific (until 60 ms) and aspecific (after 60 ms) complex of the SSEPs was delayed or the potential could not be elicited. During sleep as well as wakefulness characteristic abnormalities of the EEGs were found in nine patients. These abnormalities consisted of continuous negative sharp waves and spikes at the vertex.

Brain↗

PubMed1985

[Somatosensory chronotopography: normative data].

Somatosensory chronotopography of a limited number of components gives a simple representation of the potential distribution of evoked potentials over the skull. Components of individual somatosensory evoked potentials over 16 equally distributed electrodes were analysed in 10 healthy volunteers. The distribution of some specific and non-specific components, as found near the somatosensory cortex is defined and the distribution in relation to the individual traces plotted, resulting in seven normative topogramms with 90% appearance area of maxima of the seven different components. Dipoles are often present.

Adult↗

PubMed1984

[Neurosonology].

Explore the source record for details and available documents.

Carotid Arteries↗

PubMed1982

Autosomal dominant idiopathic hypoparathyroidism and nervous system dysfunction: report of three cases and review of the literature.

The neurological manifestations of idiopathic hypoparathyroidism in a father, his son, and his daughter are reported. In all three epilepsy was the first manifestation of the disease. Father and son also showed mental deterioration and striocerebellar symptoms; their CT scans revealed symmetrical calcification in the basal ganglia and dentate nuclei. The extent of this calcification increased during normocalcemia, which was produced by dihydrotachysterol therapy. This indicates that other factors than merely hypocalcemia influence the intracerebral calcifying process. Somatosensory evoked potentials (SSEP) showed an abnormal nonspecific complex, indicating dysfunction of the cortical gray matter. It is suggested that in the evaluation of idiopathic hypoparathyroidism one also must be beware of the possibility of epilepsy, mental deterioration, striocerebellar symptoms, intracerebral calcification and SSEP disturbances.

Adult↗

PubMed1981

Relation between EEG and disability scores in multiple sclerosis.

A disability-scoring and an EEG-spectral analysis was performed in patients suffering from clinically and biochemically (oligoclonal aspect in cerebro-spinal fluid) proven multiple sclerosis at two stages of their illness, that are before and after a short intensive immuno suppressive therapy. In both stages a relationship is found between the degree of disability and the amount of beta activity in the fronto-central area and the amount of the theta activity in the temporal area. Significant improvement in the clinical state of the patient and a marked increase of the mean alpha frequency in the parietooccipital region could be demonstrated after short intensive immunosuppressive therapy.

Adult↗

PubMed1978

Somatosensory evoked response in controlled A-alpha sensory fiber disease.

In contrast to the distal sensory response, the somatosensory evoked response (SSER) is detectable in patients with A-alpha sensory fiber diseases such as Friedreich's ataxia. In four patients with this disease a combined histological and electro-physiological investigation of sensory propagation was performed. The sural nerve was analyzed in terms of the various fiber diameters and the distal sensory and somatosensory evoked cortical responses recorded after sural and median nerve (finger) stimulation. A distinct evoked response could be detected with rather low amplitudes and retarded latency times, while in general no distal sensory response was found. The anatomical physiological consequences are discussed.

Adult↗

PubMed1978

Changes in carotid flow velocity induced by lowering cerebrospinal fluid pressure in normal pressure hydrocephalus.

This is a report of changes in blood flow velocity in the carotid system induced by lumbar puncture in five patients who had clinical and neuroradiological signs of normal pressure hydrocephalus. After lowering cerebrospinal fluid pressure an increase of carotid flow velocity was found on Doppler hematotachography. These changes of carotid blood flow velocity could not be demonstrated in a control group of four patients with normal cerebrospinal fluid dynamics. This method is easy to perform, does not overtax the patient and seems to be indicated for the diagnosis of NPH. It is presented as a new, simple, reproducible aid to the diagnosis of NPH by Doppler hematotachography.

Adult↗

PubMed1976

Changes in carotid flow velocity induced by direct percutaneous carotid angiography in man.

Quantitative percutaneous flow velocity measurements are possible in carotid arteries. The results of the direct percutaneous angiography were measured by means of a Doppler directional flow velocity device and registered on a polygraphy. The results indicate an increase in the flow velocity of the injected as well as the non-injected carotid system. This implies that there are no local constrictions of the carotid artery following intervention and, furthermore, it implies a systematic effect of the puncture and injection itself, or of the contrast medium, on the whole cerebral circulation. A diffuse vasodilatation, at least in the cerebral circulation, is postulated. The possible implications are discussed.

Adult↗

PubMed1976

Changes in carotid flow velocity during catheterization of the aortic arch and common carotid artery.

Quantitative percutaneous flow velocity measurements are possible in carotid arteries by means of a directional Doppler flow velocity device and registration on a polygraph. Carotid flow velocity changes were recorded during catheterization of the ascending aorta and common carotid artery and after infusion of 20 cc contrast medium in the ascending aorta. The results indicate an increase in the flow velocity in the right common carotid artery after infusion of the contrast medium in the ascending aorta, which is thought to be attributed to a diffuse vasodilatation, at least in the cerebral circulation. The catheterization of the ascending aorta and common carotid artery seems to interfere with the common carotid flow velocity.

Adolescent↗

PubMedDate not supplied

Peroneus brevis tendon in normal subjects: MR morphology and its relationship to longitudinal tears.

PURPOSE: The most prevalent, yet unproven, theory for the development of longitudinal splits of the peroneus brevis tendon is the compression of the peroneus brevis tendon by the peroneus longus tendon in dorsiflexion. The goal of our study was to provide insight into this pathomechanism by evaluating the shape of the peroneus brevis tendon and its relationship to the adjacent structures in the fibular groove during plantarflexion and dorsiflexion. METHOD: The MR images of 13 ankles in asymptomatic adult volunteers were performed in full dorsiflexion and plantarflexion. The axial MR images were assessed for the shape of the peroneus brevis tendon and its relationship to the peroneus longus tendon and posterior cortex of the fibula in both plantarflexion and dorsiflexion. RESULTS: In 12 of the 13 volunteers, the peroneus brevis tendon was located anterior or anteromedial to the peroneus longus tendon in the fibular groove. In those volunteers the peroneus brevis tendon was more flattened and compressed against the fibular groove by the overlying peroneus longus tendon in dorsiflexion than plantarflexion. Fat planes were noted in plantarflexion between the peroneal tendons as well as between the peroneus brevis tendon and the fibular groove. These were obliterated in dorsiflexion. CONCLUSION: The changes in configuration of the tendon of the peroneus brevis tendon in dorsiflexion compared with plantarflexion provide support to our present understanding of the pathomechanism of longitudinal tears of the peroneus brevis tendon.

Adult↗

PubMedDate not supplied

Vascular compression in the thoracic outlet. Age dependent normative values in noninvasive testing.

Thoracic outlet syndromes are the result of vascular and/or nerve compression. In order to diagnose these syndromes careful clinical examination and objective testing are required. Vascular compression can be provoked by three well-defined thoracic outlet compression manoeuvres. Changes in vascular pulsation during these manoeuvres can be registered by means of plethysmography of the fingers. For an adequate interpretation of the clinical value of the various techniques of provoking vascular compression, a knowledge is required of the frequency with which vascular compression can be provoked in normal subjects of different age groups. We measured the changes in pulse in normal volunteers in 5-year age groups ranging from 15 to 85. This study showed that vascular compression until zero flow during 120 degrees arm abduction seems to be normal rather than abnormal. Zero flow during Adson manoeuvre and military attitude did not occur frequently in the supine position in normal subjects. Neither the different age groups nor the sexes showed any real differences in the quality of the findings. Therefore, establishing zero flow by means of a costoclavicular compression or scalenic compression is a clinically significant finding, while zero flow during 120 degrees abduction (pectoralis minor compression) is within normal limits.

Adolescent↗