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Biomedical subjects

E B Werner

Publications and source records attributed to E B Werner.

32 records · Page 2Linked to original sources
PubMed1983

Relationship between baring of circumlinear vessels of the optic disc and glaucomatous visual field loss.

Stereo disc photographs of the eyes of 232 patients with normal or elevated intraocular pressure were reviewed to assess the value of baring of circumlinear vessels of the optic nerve head as a sign of glaucomatous damage. Baring and the presence of glaucomatous visual field defects were significantly correlated, as were this sign and consistent elevation of the intraocular pressure in patients without a visual field defect. Baring was absent in 35% of the eyes with circumlinear vessels and known glaucomatous visual field defects. However, it was present in only 6% of the eyes with normal pressure, a normal visual field and circumlinear vessels. Therefore, this sign should lead to further evaluation.

Adult↗
PubMed1982

Variability of static visual threshold responses in patients with elevated IOPs.

We determined the static visual thresholds within 15 degrees of fixation of 36 patients with elevated intraocular pressures but normal visual fields and compared the result with 36 controls matched for age and sex. We found no difference in the mean threshold level between the two groups. We did find a tendency for the patients with elevated IOPs to have greater variability or scatter of their threshold responses close to fixation. We also found the patients had larger cup to disc ratios than the controls.

Humans↗
PubMed1982

Risk factors for progression of visual field defects in medically treated patients with glaucoma.

A multivariate discriminant analysis of potential risk factors for the progression of visual field defects was performed in 70 patients with chronic open-angle or low-tension glaucoma. Male sex, low diastolic blood pressure and diabetes mellitus were found to be significant risk factors. When the patients with low-tension glaucoma were excluded, a high mean intraocular pressure was identified as a risk factor. It was concluded that there is no reliable way to predict future visual field loss in patients with glaucoma.

Blood Pressure↗
PubMed1980

Temporal visual field defects in glaucoma.

In a study of 151 glaucomatous eyes with typical nerve fibre bundle defects we found that 4 (3%) had isolated temporal visual field defects. We stress the importance of studying the temporal field in patients with glaucoma.

Aged↗
PubMed1980

Location of early glaucomatous visual field defects.

A study of 50 eyes with chronic open-angle glaucoma and early visual field defects showed superior defects to be twice as common as inferior defects. The superior defects tended to be closer to fixation than the inferior defects.

Glaucoma↗
PubMed1979

Peripheral nasal field defects in glaucoma.

One hundred fifty-one eyes of 101 consecutive patients with chronic open-angle and low tension glaucoma showed typical visual field changes. Sixty of the eyes had a nasal step, either alone or in combination with other defects. In 17 of these eyes, it was possible to demonstrate an isolated scotoma in the nasal periphery. It was concluded that the peripheral nasal step is a nerve fiber bundle defect that in its earliest phase produces a scotoma. In this sense, it behaves similar to the more central defects, rather than simply as a depression of isopters nasally.

Adult↗
PubMed1977

Early visual field disturbances in glaucoma.

Twenty-two eyes of 22 patients with initially normaly visual fields developed glaucomatous field defects. In 13 of these, the development of the definitive field defect was preceded by a localized minor disturbance in the area where the defect appeared subsequently. In a control group of 22 ocular hypertensives without field defects, six eyes had localized disturbances similar to those that preceded the field defects in the study group. This type of minor localized disturbance may represent an early glaucomatous visual change detectable before the appearance of definitive scotomas.

Aged↗
PubMed1977

Trabeculectomy and the progression of glaucomatous visual field loss.

Twenty-four eyes of 20 patients with chronic open angle glaucoma who had a trabeculectomy were studied retrospectively. Trabeculectomy resulted in a significant lowering of mean intraocular pressures and improvement of pressure control for the group. Progression of visual field defects occurred in all 24 eyes preoperatively, while postoperatively, the fields of 14 eyes showed no change, but the other ten showed further progression. A comparison of the pressures of the eyes showing continued progression after surgery with the eyes showing no further progression revealed no significant difference in the mean preoperative or postoperative pressure or the mean pressure reduction after surgery. Measurements of the quality of the control, however, showed that the quality of pressure control was significantly better in the eyes that did not show progression of field loss postoperatively. Careful postoperative follow-up observation of the visual field remains necessary even after successful surgical pressure reduction.

Adult↗
PubMed1977

Increased scatter of responses as a precursor of visual field changes in glaucoma.

A patient with ocular hypertension developed a glaucomatous field defect in the right eye 10 years after the initial examination. For several years preceding the appearance of the defect there had been scatter of threshold responses localized to the area where the defect was later found. We suggest that localized scatter may be an early sign of glaucomatous damage to visual function.

Aged↗
PubMed1977

Progression of glaucomatous field defects despite successful filtration.

It is generally assumed that successful filtering surgery, by maintaining intraocular pressure at a low level, will protect a glaucoma patient's remaining visual field. We present three patients with chronic open angle glaucoma and typical visual field changes. In each case, filtering surgery was performed because of progressive loss of visual filed and inadequate pressure control. Despite excellent pressure levels after operation, field loss continued in the operated eye. Certain genetic and systemic factors may contribute to further loss of visual field in the presence of normal intraocular pressures. We stress the importance of careful and continuous examination of the visual fields in all glaucoma patients regardless of the response of pressure to treatment.

Adult↗