Biomedical subjects
D C Haas
Publications and source records attributed to D C Haas.
Prolonged migraine aura status.
Explore the source record for details and available documents.
Carpal tunnel syndrome following automobile collisions.
Seven patients developed symptoms and signs of carpal tunnel syndrome after vehicles they were driving were involved in collisions. Nerve conduction studies showed median nerve impairment at the wrist in 4 patients and suggested it in the other three. The presence of carpal tunnel syndrome was confirmed in every patient by the relief of symptoms following surgical division of the volar carpal ligament. The findings suggest that injuries inflicted on nonneural structures of the carpal tunnel when the patient was gripping the steering wheel led to chronic compression of the median nerve within the tunnel.
Treatment of polymyositis with niridazole.
Explore the source record for details and available documents.
Graves' disease, myasthenia gravis, and purpura.
Explore the source record for details and available documents.
Vitamin E therapy in polymyositis.
Explore the source record for details and available documents.
Juvenile head trauma syndromes and their relationship to migraine.
The clinical spectrum of juvenile head trauma syndromes was derived from an analysis of 50 attacks in 25 patients. Attacks were grouped into four clinical types: (1) hemiparesis; (2) somnolence, irritability, and vomiting; (3) blindness; and (4) brain stem signs. Our evidence shows that these four types are different manifestations of a common underlying process. All attacks followed mild head trauma after a latent interval, generally of one to ten minutes. Forty of the 50 attacks occurred in patients under 14 years of age. Full recovery occurred after a variable time in all but one instance. This patient, and one other, had an angiographically demonstrable occlusion of a branch of the middle cerebral artery. In clinical and laboratory features, these attacks resemble classical migraine and presumably have a similar underlying mechanism.
Cell-mediated immunity in polymyositis. Creatine phosphokinase release from muscle cultures.
Explore the source record for details and available documents.
Treatment of polymyositis with immunosuppressive drugs.
Explore the source record for details and available documents.
On a possible relationship of cyclic AMP to the mechanism of action of nerve growth factor.
Explore the source record for details and available documents.
Animal model for human disease: Idiopathic polyneuritis, Guillain-Barré Syndrome. Animal model: Coonhound paralysis, idiopathic polyradiculoneuritis of coonhounds.
Explore the source record for details and available documents.
Migraine attacks triggered by mild head trauma, and their relation to certain post-traumatic disorders of childhood.
Explore the source record for details and available documents.
Neurological effects of glutethimide.
Explore the source record for details and available documents.
Coonhound paralysis. An acute idiopathic polyradiculoneuritis in dogs resembling the Landry-Guillain-Barré syndrome.
Explore the source record for details and available documents.
Absence of cell-mediated cytotoxicity to muscle cultures in polymyositis.
Blood mononuclear cells from 10 patients with polymyositis and 22 controls were incubated with fetal-rat muscle cultures. Polymyositis cells produced no significant cytotoxic effects in cultures as determined by visual inspection and measurement of the release from cultures of creatine phosphokinase and radioactive chromium. These negative results contrast with previous reports and raise the question of whether blood mononuclear cells from polymyositis patients are cytotoxic for muscle cultures.