Enhancement of the locomotor response to d-amphetamine by olfactory bulb damage in rats.
Explore the source record for details and available documents.
Animals↗
Biomedical subjects
D B Neill
Publications and source records attributed to D B Neill.
Deficits in consummatory responses to regulatory challenges following basal ganglialesions in rats.
Explore the source record for details and available documents.
Animals↗
Comparison of the effects of frontal, striatal, and septal lesions in paradigms thought to measure incentive motivation or behavioral inhibition.
Explore the source record for details and available documents.
Animals↗
Behavioral effects of amphetamine in rats with lesions in the corpus striatum.
Explore the source record for details and available documents.
Amphetamine↗
Selective potentiation of locomotor effects of amphetamine by midbrain raphé lesions.
Explore the source record for details and available documents.
Amphetamine↗
Interaction of the effects of reserpine and amphetamine on food and water intake.
Explore the source record for details and available documents.
Amphetamine↗
Behavioral effects of lesions or cholinergic blockade of the dorsal and ventral caudate of rats.
Explore the source record for details and available documents.
Animals↗
Amphetamine reversal of sexual impairment following anterior hypothalamic lesions in female rats.
Explore the source record for details and available documents.
Animals↗
Effects of intrastriatal injections of scopolamine on appetitive behavior.
Explore the source record for details and available documents.
Amphetamine↗
Effects of lesions in the dorsal or ventral striatum on locomotor activity and on locomotor effects of amphetamine.
Explore the source record for details and available documents.
Amphetamine↗
Impairment of avoidance performance by intrastriatal administration of 6-hydroxydopamine.
Explore the source record for details and available documents.
Animals↗
Striatal dopaminergic modulation of lateral hypothalamic self-stimulation.
The bilateral application of crystalline 6-hydroxydopamine to the ventral anterior head of the corpus striatum of rats severely suppressed responding for electrical stimulation of the lateral hypothalamus. This suppression lasted for days or weeks, after other behavioral deficits in food and water intake and activity had recovered. Application of crystalline dopamine through the same cannulas temporarily reversed the impairment. Applications of crystalline dopamine to the same striatal region of untreated rats were more effective in enhancing self-stimulation than similar applications of norepinephrine.
Animals↗