Biomedical subjects
Publications and source records attributed to C Soriano.
Traditional and current concepts, anatomical and physiological mechanisms, causes, classification, clinical manifestations, diagnostic approach, and current therapy of hemorrhoidal disease are reviewed in this article. It is encouraged to use the term hemorrhoidal disease instead of the misused one hemorrhoids.
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Despite routine monitoring, a number of prolonged apneic and bradycardic episodes were undetected in a group of infants in the neonatal intensive care unit (NICU). Sixty-one infants were evaluated by 12-hour pneumocardiograms at a post-conceptional age of 35 +/- 3 (SD) weeks. Nursing documentation failed to detect 11 infants with prolonged apnea and bradycardia. Three of these infants were not detected in spite of increased awareness following in-service education. Such a lack of documentation may lead to improper medical management of infants at risk for pathologic apnea and suggests the need for more accurate documentation at the time of discharge.
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BACKGROUND: A preceding viral syndrome is classically cited as a triggering factor for thrombotic microangiopathy although only anecdotal reports of the causal agent have been documented. CASE REPORT: An 84-year-old woman developed thrombotic microangiopathy secondary to diarrhea. An exhaustive etiological work-up documented the presence of Rotavirus alone in the fecal matter. The clinical course was favorable after 9 plasma exchanges using frozen fresh plasma and was associated with disappearance of the Rotavirus. DISCUSSION: Enterohemorrhagic Escherichia coli is the most commonly identified infectious agent causing thrombotic microangiopathy. A small number of cases caused by viral agents (particularly retroviruses) have also been documented. To our knowledge, this is the first case attributable to a Rotavirus. In our case, the presence of the Rotavirus in the fecal matter and the favorable clinical course when the virus disappeared would be in favor of its causal role in the pathogenesis of thrombotic micorangiopathy.