Exposure of rabbits to 50 ppm carbonyl sulfide. A biochemical and histomorphological study.
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Animals↗
Biomedical subjects
C Hugod
Publications and source records attributed to C Hugod.
Temporal arteritis--progressive affection of vision during high-level corticosteroid therapy. A case report.
A case of giant cell arteritis (temporal arteritis) is presented in a 73-year-old male patient in whom lasting reduction of vision developed during high-level prednisone treatment (60 mg daily) at a time when clinical improvement in other respects had appeared.
Aged↗
[Passive smoking--exposure to the gaseous phase and the particle phase of cigarette smoke].
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Acrolein↗
Exposure of passive smokers to tobacco smoke constituents.
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Air Pollution↗
[Giant cell arteritis of the ovary].
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Aged↗
Effect of carbon monoxide exposure on aortic and coronary intimal morphology in the rabbit. A revaluation.
Non-cholesterol-fed rabbits were exposed to carbon monoxide at concentrations in air of either 200, 2000, or 4000 parts per million (=0.02, 0.2 or 0.4%, vol/vol). Using the same criteria for intimal damage as in earlier morphological studies, no histotoxic effect on intimal/subintimal morphology of coronary arteries or the aorta could be demonstrated, when light-microscopic evaluation was performed blindly.
Animals↗
[Rheumatic polymyalgia--temporal arteritis. Temporary remission without glucocorticoid therapy].
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Aged↗
[Strangulation by main entrance doors. 2 identical accidents in which preschool children were strangled].
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Accident Prevention↗
[The value of the intravenous galactose tolerance test and serum vitamin B 12 level in the differential diagnosis and prognosis of liver diseases].
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Acute Disease↗
Ultrastructural changes of the rabbit lung after a 5 ppm nitric oxide exposure.
Four male rabbits were continuously exposed to 5 ppm nitric oxide for 14 days. Four rabbits served as controls. Specimens from pulmonary arterioles and alveolar tissue were examined by light and transmission electron microscopy. The most conspicuous findings were fluid-containing vacuoles inside the arteriolar endothelial cells and/or in the intercellular junctions. Thickening of the alveolo-capillary membrane due to accumulation of edema fluid in the interstitial space was also observed.
Animals↗