THE TRAINING OF THE PHYSICIAN. THE RESIDENCY. II.
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Hospitals↗
Biomedical subjects
C H BURNETT
Publications and source records attributed to C H BURNETT.
A GENETIC STUDY OF HEREDITARY OROTIC ACIDURIA.
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Anemia↗
VITAMIN D-RESISTANT RICKETS. ANALYSIS OF TWENTY-FOUR PEDIGREES WITH HEREDITARY AND SPORADIC CASES.
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Blood Chemical Analysis↗
Changing trends in clinical investigation.
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Humans↗
Changing trends in clinical investigation.
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Humans↗
An analysis of some features of renal tubular dysfunction.
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Humans↗
A genetic study of familial hypophosphatemia and vitamin D resistant rickets with a review of the literature.
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Heredity↗
A genetic study of familial hypophosphatemia and vitamin D resistant rickets.
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Familial Hypophosphatemic Rickets↗
Blood volume and extracellular fluid volume during administration of ACTH and cortisone.
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Adrenocorticotropic Hormone↗
The effects of venous congestion of the limbs upon renal clearances and the excretion of water and salt. I. Studies in normal subjects and in hypertensive patients before and after splanchnicectomy.
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Extremities↗
Observations on the electrolyte and water metabolism in Addison's disease during oral salt loading.
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Addison Disease↗
The effects of ACTH and cortisone on the renal tubular transport of uric acid, phosphorus, and electrolytes in patients with normal renal and adrenal function.
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Adrenocorticotropic Hormone↗
The effects of cortisone on the course of acute glomerulonephritis; report of a case.
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Acute Disease↗
Extensive laboratory studies of a patient with pheochromocytoma before and after successful operation; with a note on the trial of piperidylmethyl benzodioxane to differentiate such conditions from essential hypertensive vascular disease.
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Adrenal Glands↗
Studies of alkalosis; renal function during and following alkalosis resulting from pyloric obstruction.
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Alkalosis↗
Studies of alkalosis; electrolyte abnormalities in alkalosis resulting from pyloric obstruction.
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Alkalosis↗
Panhypopituitarism: case report correlating clinical and pathologic findings.
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Humans↗
Changes in normal renal function resulting from ACTH and cortisone.
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Adrenocorticotropic Hormone↗