Congenital bronchoesophageal fistula in an adult: a case presenting with massive hemoptysis.
Explore the source record for details and available documents.
Biomedical subjects
Publications and source records attributed to C E Kinley.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
An increasing interest in educational science as applied to medical education is apparent in the past decade. Recently, a six-week teacher training program was held at the Center for the Study of Medical Education at the University of Illinois. Fifteen medical faculty members, including five Canadians, participated. During this period there was an opportunity to engage in discussions and independent study of several aspects of medical education, using the personnel from the Center's four major divisions as resource people. Training in educational science is important for all teachers, and a centre to provide this type of instruction should be available in Canada.
Explore the source record for details and available documents.
Compliance mismatch between host artery and synthetic vascular substitutes has rarely been mentioned in the literature as a possible cause of suture line disruption and damage to endothelial and platelet cells. In this paper we have attempted to elucidate on how compliance mismatch can predispose: (1) anastomotic rupture, (2) flow induced shear stres and, (3) reduced distal perfusion. We have concluded that in order to minimize all three of these factors the compliance of the vascular substitute should be equivalent to the host artery to which it is to be anastomosed. It appears that the factor which is most seriously affected by the compliance mismatch is the flow regimen in that flow separation regions forms at the distal anastomosis. Shear stresses, due to eddies in these regions of flow separation, are of significant magnitude to destroy endothelial cells lining the vascular wall and platelets existing in the boundary layer.