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Biomedical subjects

C Dahl

Publications and source records attributed to C Dahl.

84 records · Page 5Linked to original sources
PubMed1984

Leydig cell tumour--a malignant tumour?

A case of feminizing Leydig cell tumour is presented. The report includes the results of paraclinical tests performed inter alia to determine whether this rare tumour was benign or malignant.

Adult↗
PubMed1983

Do thiazides prevent recurrent idiopathic renal calcium oxalate stones?

In a double-blind controlled clinical trial 62 patients with recurrent idiopathic renal calcium oxalate stone formation were allocated either to treatment with bendroflumethiazide, 2.5 mg three times a day, or placebo. In each group the rate of stone formation during medication (average follow-up period 36 months) was compared with the rate of stone formation before medication (average control period 36 months). In both groups a similar striking fall in the rate of stone formation was found, indicating that thiazides in this study did not alter the spontaneous course of idiopathic renal calcium oxalate stone formation. It is doubtful whether life-long prophylaxis with thiazide is justified in patients with a moderate rate of stone formation.

Adolescent↗
PubMed1982

Effect of quinidine on the heart rate and blood pressure response to treadmill exercise.

The effect of quinidine on the heart rate and blood pressure response to treadmill exercise was evaluated in 17 normal subjects. Quinidine significantly increased the heart rate at rest and at low levels of exercise. Quinidine had no significant effect on the systolic blood pressure response to any work load. However, quinidine significantly decreased the systolic blood pressure at all exercising heart rates, compared to control, in 15 of the 17 subjects. The magnitude of the decrease in systolic pressure ranged from 1 to 22 mm Hg, with a mean of 10 mm Hg.

Adrenergic alpha-Antagonists↗
PubMed1975

Myocardial injury from transthoracic defibrillator countershock.

Myocardial damage resulting from transthoracic administration of direct current with cardiac defilbrillators present in clinical use was studied in 66 dogs. Electrodes were applied to the thoracic skin. All direct current discharges were delivered via a commercially available defilbrillator. Most of the animals received ten consecutive discharges with a dial setting of 400 watt-seconds. Animals were killed from 3 to 14 days after receiving the discharges. Myocarial necrosis was produced in most of the animals. The lesions were characterized by sharply localized areas of muscle necrosis that progressed to fibrous scars. Mineralization of damaged muscle and florid proliferation of large mononuclear cells were striking features of the lesions.

Animals↗