Biomedical subjects
C A Pickering
Publications and source records attributed to C A Pickering.
Asthma in workers manufacturing cephalosporins.
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A 7 year survey of 'allergic' occupational lung disease in the United Kingdom.
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"Coughing water": bronchial hyperreactivity induced by swimming in a chlorinated pool.
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Respiratory allergy to a factory humidifier contaminant presenting as pyrexia of undertermined origin.
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Extrinsic allergic alveolitis and contaminated cooling-water in a factory machine.
Symptoms and signs typical of extrinsic allergic alveolitis occurred in 24 workers in a stationery factory. The illness was caused by inhalation of a water aerosol contaminated by microorganisms, and although no specific organism has been incriminated, affected workers had serum precipitins to the contaminated water, and the illness could be reproduced by inhalation challenge tests.
Nocturnal asthma and urinary adrenaline and noradrenaline excretion.
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Bronchial provocation tests [proceedings].
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Investigation of a respiratory disease associated with an air-conditioning system.
This paper describes the investigation of respiratory disease presenting in a group of men employed on the printing floor of a factory. The air-conditioning units supplying this floor were found to be contaminated with a variety of micro-organisms. The specific cause of the disease was not identified but an alteration in the system of humidification led to the complete resolution of symptoms.
Letter: Mushroom worker's lung.
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Asthma due to inhaled chemical agents--piperazine dihydrochloride.
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Asthma due to inhaled chemical fumes--amino-ethyl ethanolamine in aluminium soldering flux.
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Asthma due to inhaled wood dusts, Western red cedar and Iroko.
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Asthma due to inhaled chemical agents, tolylene di-isocyanate.
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Asthma due to inhaled chemical agents--complex salts of platinum.
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Inhalation tests with chemical allergens: complex salts of platinum.
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Pulmonary capillary haemangiomatosis.
A Cypriot male of 22 years died after suffering recurrent daily haemoptyses of varying severity and slowly increasing effort intolerance for a period of four and a half years. Examination of a lung biopsy specimen revealed dilated vascular channels suggestive of a congenital anomaly of the pulmonary vasculature. Histological examination of the lung following necropsy revealed the diagnosis of pulmonary capillary haemangiomatosis. In this condition sheets of thin-walled blood vessels infiltrate the lung parenchyma, the walls of pulmonary arteries and veins, the bronchi and the pleura. The infiltration of pulmonary veins and venules induces secondary pulmonary veno-occlusive disease. This appears to be the second example of this disease which has been reported. It is open to clinical and histological mis-diagnosis and clinicians and pathologists should be aware of the existence of this entity.