Biomedical subjects

B Sureda

Publications and source records attributed to B Sureda.

18 recordsLinked to original sources

PubMed1991

[Pregnancy and benign intracranial hypertension].

Several papers have suggested that pregnancy is one of the etiopathogenic factors of benign intracranial hypertension (BIH). The therapeutic attitude to be taken as regards new pregnancies in women previously afflicted with BIH during pregnancy is still on discussion. This paper is based on a study of 100 BIH cases. The results support the idea that cases, but the obesity involved. The coexistence of BIH and pregnancy does not increase the risk of relapse, does not mean a worse prognosis of BIH nor does it appear to have a negative effect on the child. Any woman who has previously developed BIH during pregnancy should not be advised against future pregnancies. In the case of a new pregnancy, a very close control should be carried out in order to avoid an excessive increase in weight.

Adult↗

PubMed1991

[Recurrence of benign intracranial hypertension].

The recurrence of benign intracranial hypertension (BIH) in 100 patients was analysed after a long-term follow-up. A recurrence appeared in 20% of the cases in this series. This being more frequent, with statistical significance, in females aged between 20-40 years. Obesity was the more frequent etiopathogenic factor involved in the onset of relapses. Other factors were pregnancy, steroid therapy, levothyroxine and obstruction of the cerebrospinal fluid derivation system. Relapses did not produce loss of sight. We insist on the elimination of developing factors in order to prevent the recurrence.

Adolescent↗

PubMed1990

[Benign intracranial hypertension and obesity].

The incidence of obesity and excess weight were analyzed in a series of 100 cases of benign intracranial hypertension (BIH) and compared with a control group. The results of hypocaloric diet and lumbar puncture were compared to those obtained with other treatments. Relapses of the disease were analyzed in obese patients who maintained a normal weight after clinical recovery and compared with those who get fat again. A similar analysis was carried out in pregnant women. The frequency of sequelae in obese patients was also determined. A high incidence of obesity was observed only in women aged 20 to 40 years who presented BIH. Hypocaloric diet and lumbar puncture constituted the treatment which exerted a more marked shortening of the clinical course. Relapses of BIH were more frequent in obese patients who did not normalize the body weight and in new pregnant women. Data indicate the relevance of obesity as etiopathogenetic factor in a group of patients with BIH.

Adolescent↗

PubMed1990

[Recurrent hypersomnia caused by acute toluene poisoning].

Explore the source record for details and available documents.

Acute Disease↗

PubMed1989

Cyst of the septum pellucidum and Korsakoff's psychosis.

Noncommunicating cysts of the septum pelludicum are rare and diagnostic experience with CT scan is scanty. They may cause hydrocephalus because of obstruction of Monro's foramina. In this report we describe a young patient with acute Korsakoff's syndrome. The CT scan revealed a cyst of the septum pellucidum without hydrocephalus. Surgery disclosed a noncommunicating cyst compressing structures of the limbic system. After surgery symptoms disappeared.

Adult↗

PubMedDate not supplied

[Visual evoked potentials in benign intracranial hypertension].

In 20 patients with benign intracranial hypertension (BIH) a study of the P100 latency of visual evoked potentials (VEP) was carried out. At admission only 5 patients showed abnormal latencies. The mean of the P100 latencies in patients was significantly increased as compared with controls. Latencies persisted relatively prolonged once the funduscopy had become normal. In the last investigation, one year after BIH had disappeared, the latencies in patients were already similar to those in the control group. On the other hand, no patient with normal latencies had visual sequelae. Sequelae also did not develop in patients in whom initially prolonged latencies progressively returned to normal during the evolution. Sequelae developed in one patient in whom latencies became progressively prolonged. In that case the increased latencies preceded the fall in visual acuity. These data may be useful to evaluate visual function in patients with BIH.

Evoked Potentials, Visual↗

PubMedDate not supplied

[Episodic ++paroxysmal hemicrania or chronic paroxysmal hemicrania in pre-chronic state?].

Two women suffered from strictly unilateral, periocular headaches for many years. The clinical characteristics of headaches were those described in chronic paroxysmal hemicrania. However, both patients showed an episodic evolution of pain. In one case, this stage could be considered pre-chronic because it was later followed by the classic and apparently definitive chronic period of the disorder. In the second case the episodic stage persisted all along the evolution of the disease and the headache did not reach the chronic period. The patient has been free of pain without treatment for the last three years. This second observation can be considered as an example of episodic paroxysmal hemicrania. In the episodic cases the response of pain to indomethacin can be difficult to be ascertained.

Adult↗

PubMedDate not supplied

[Pituitary study in benign intracranial hypertension].

Pituitary function studies were performed on 20 patients with benign intracranial hypertension. Abnormal results were only obtained in six cases and all of these findings could be attributed to sources other than the increased intracranial pressure. Once the disease as well as its cause had disappeared, a second study was carried out and the results for all the patients were then normal. Computerized tomographic scanning of the sella turcica detected an empty sella in one patient and a possible hypophyseal microadenoma without functional repercussions in another. No evidence was found that benign intracranial hypertension is provoked by an impairment in pituitary function, nor that it could be a secondary complication thereof.

Empty Sella Syndrome↗

PubMedDate not supplied

[Prophylactic treatment of episodic cluster headaches with methysergide and prednisone].

In two consecutive series of 25 cases each suffering from episodic cluster headache, one of the clusters was treated prophylactically in the first series with methysergide and in the other with prednisone. The duration of the clusters under treatment was similar in both series. The results of our study suggest that neither the methysergide nor the prednisone were able to shorten the duration of clusters.

Adolescent↗

PubMedDate not supplied