Biomedical subjects
Publications and source records attributed to B J Ansink.
The case of a 63-year old man is presented in whom remission of a primary central nervous system lymphoma was achieved by corticosteroids only. After discontinuation of steroid therapy the remission persisted for two and a half years. Recurrences appeared at other sites of the brain, and were steroid resistant.
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Despite the recent report of Goldstein et al. (1975) on pure word deafness in an adult, it still seems worthwhile to study the case of cortical deafness in a child, not only because we believe this to be one of the very few cases duly reported upon, but also because of its interesting pathological history, its implications for the existence of specific speech sound detectors, and its evidence of pathological communicative behavior which deviates from that of a deaf child.
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In 28 patients suffering from subacute combined degeneration of the spinal cord, vitamin B12 metabolism was investigated. Two postulates, proving vitamin B12 deficiency and excluding another cause for the clinical symptoms, have to be fulfilled. Two patients had no disturbance in their vitamin B12 metabolism. Seven patients had a distinct vitamin B12 deficiency. In the remaining 19 patients we found a mild vitamin B12 deficiency. Of these patients, 5 had had a subtotal gastrectomy, one had had a low absorption of vitamin B12, and 13 patients we could not find a distinct cause for the vitamin B12 deficiency. It is not impossible that nutritional habits can be hold responsible for this deficiency. The question whether these 13 patients should be treated with vitamin B12 for the rest of their lives is difficult to answer. It is a conditio sine qua non that in the patients with S.C.D. the vitamin B12 metabolism is examined circumstantially. By so doing, it may be possible to detect, in cases with minor clinical signs and symptoms of S.C.D., the cause of their illness.
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Explore the source record for details and available documents.
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Overall motor retardation and clumsiness; bilateral pyramidal signs; extensor plantar responses; extra pyramidal symptoms; central hearing loss; perception disorders almost exclusively for linguistic items; diffuse E.E.G. abnormalities with focal maxima in both temporal regions.