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Biomedical subjects

B Dalens

Publications and source records attributed to B Dalens.

83 records · Page 5Linked to original sources
PubMed1979

[Meckel's syndrome: an unusual pedigree].

The authors report the case, in the same family, of two children with the Meckel's syndrome, of two mis-arriages (one foetus with anencephalia, the other one with occipital meningocele), and of two still-born children (one with anencephalia, the other one with occipital meningocele). The genetic problems raised by this unusual genealogy are discussed.

Anencephaly↗
PubMed1978

Cerebrospinal fluid enzymology: creatine kinase, lactate dehydrogenase activity and isozyme pattern as a brain damage index.

Blood serum and cerebrospinal fluid from a 4.5-year-old girl suffering from convulsive episodes of toxic origin were investigated for lactate dehydrogenase (LDH) and creatine kinase (CK) activities. Elevated levels of both enzymes were found. Furthermore CK was higher in the CSF (680 I.U./1) than in blood serum (160 I.U./1). The CSF activity was demonstrated mainly as the BB form (96%) for CK and H4 (63%) was the predominant form for LDH. Identical investigations were performed 45 h later and results compared with the first set. These data provide an additional example of interest in CSF enzymatic studies as a brain damage index.

Alcoholic Intoxication↗
PubMedDate not supplied

[Extramembranous glomerulonephritis during congenital syphilis].

A 3 month old girl with congenital syphilis presented with the nephrotic syndrome due to extra membranous glomerulonephritis. The presence of the first component of the classical pathway of complement activation (C1q) in the glomerular deposits and the antitreponemal antibodies in circulating cryoglobulins (titre 1 : 320) support the hypothesis that immune complexes are responsbile for the glomerulonephritis in congenital syphilis.

Antibodies, Bacterial↗
PubMedDate not supplied

Cerebrospinal fluid cytomorphology in neonates.

A prospective cytologic study of cerebrospinal fluids from 57 full-term neonates confirmed the previously reported cellular polymorphism. The cells were classified as (1) exogenous cells, (2) blood and bone-marrow cells, (3) histiomonocytes, (4) cell aggregates and (5) "free" cells. Histiomonocytes were a constant feature in these fluids and were the cause of neonatal pleiocytosis. Various degrees of activation could be recognized. The so-called inactive forms were closely related to brain damage. Identification of erythrophages and siderophages had diagnostic significance. The presence of fat in macrophages (lipophages) suggested a serious developmental disorder. The free cells were incompletely identified, as were the cell aggregates, which probably originate in the meningeal lining. Both require further study.

Bone Marrow Cells↗