[Accident and insurance medicine].
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Abdominal Injuries↗
Biomedical subjects
A Wilhelm
Publications and source records attributed to A Wilhelm.
[Subcutaneous arteriovenous shunt for intermittent hemodialysis treatment].
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Arteriovenous Anastomosis↗
[Accident and insurance medicine].
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Accidents↗
[10. Denervation of the wrist].
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Denervation↗
[On th etiology, diagnosis and treatment of unclear pain in the wrist].
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Adolescent↗
[Care of skin injuries of the hand].
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Bone Transplantation↗
[Injuries of the extension aponeurosis on the end phalanx of the finger and their treatment].
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Calcium Sulfate↗
[1b. The subcutaneous arteriovenous fistula for cannulation in intermittent hemodialysis].
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Humans↗
[On the treatment of intra-articular fractures of the hand].
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Finger Injuries↗
A subcutaneous arteriovenous fistula for use in intermittent haemodialysis.
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Humans↗
[Accident and health insurance].
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Germany, West↗
[Articular denervation and its anatomical foundation. A new therapeutic principle in hand surgery. On the treatment of the later stages of lunatomalacia and navicular pseudarthrosis].
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Bibliographies as Topic↗
[Accident-induced simultaneous occurrence of a chylothorax and of a subphrenic chyle pseudocyst].
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Accidents↗
[Accident and insurance medicine].
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Humans↗
Regulation of luteinizing hormone secretion in prepubertal male and female rats.
The effect of naloxone or clonidine, on serum levels of luteinizing hormone (LH) was investigated in immature male and female rats. After birth, naloxone progressively increased serum LH concentrations in females, reaching maximal effects on the 10th to the 16th day of life. Males failed to respond to naloxone during this period. Clonidine only moderately increased LH in immature female rats. However, immature males displayed a high sensitivity to this alpha 2-adrenoceptor agonist. The secretion pattern of LH caused by clonidine during the first two weeks of life resembled that evoked by naloxone in females. Thus, regulation of LH secretion of immature female rats involves endorphins, while in males control of LH-release is by adrenergic mechanisms.
Animals↗