Search PubMed⌕ Search

Biomedical subjects

A Vandevenne

Publications and source records attributed to A Vandevenne.

At least 37 records · Page 2Linked to original sources
PubMed1987

[Characteristics and evolutive data of chronic respiratory diseases after respiratory intensive care. Evaluation of the Pneumology Department of the Pavillon Saint-François in Strasbourg between 1980 and 1985].

Between 1980 and 1985, 66 patients with chronic obstructive lung disease (respiratory deficit of the restrictive type) were admitted to our department after an episode of acute respiratory failure treated with assisted ventilation in an intensive care unit. These patients were in a particularly poor clinical condition, due to their previous long stay in the intensive care unit (mean 43 days), the high percentage of tracheotomies (mean 44%), the loss of autonomy of movement in 30% of the cases and the presence of an associated pathology in 45% of the patients. These data explain the high mortality observed in this group: 40% of the patients died within one year of the acute respiratory failure episode. Other prognostic factors, notably the patients' nutritional status, must also be taken into account.

Aged↗
PubMed1987

[Pulmonary tuberculosis and cutaneous anergy to tuberculin].

A negative tuberculin test in a bacilliferous subject is an apparently paradoxical situation, since the first penetration of tubercle bacilli into the human body is known to induce hypersensitivity to tuberculin and to enhance cellular immunity. These two facets of cutaneous reaction are most probably related to lymphocyte populations of two different phenotypes. The authors found that 21% of skin tests were negative in patients carrying tubercle bacilli, with a higher frequency in elderly people and in subjects with low serum albumin levels and/or biochemical hepatic alterations. Helped by the literature, they are wondering about the significance and possible mechanisms of this situation.

Adult↗
PubMed1985

Flow-volume curve abnormalities and obstructive sleep apnea syndrome.

Recent reports have suggested that flow volume curve abnormalities may be of interest in the diagnosis of obstructive sleep apnea syndromes by showing either extrathoracic airway obstruction (ratio of expiratory flow to inspiratory flow at 50 percent of forced vital capacity [FEF50/FIF50] exceeding 1) or upper airway fluttering (indicated by a sawtooth aspect on the mid-half of the inspiratory part of the curve) or both. In our study, 57 patients referred for a suspected sleep apnea syndrome (SAS) underwent conventional spirometry, assessment of flow-volume curves, ENT examination, and polysomnography. Thirty patients had an obstructive SAS, four patients a central SAS, and 23 patients no SAS. Signs of upper airway fluttering (the sawtooth sign) were present in 61 percent of the patients with obstructive SAS and in 46 percent of the patients without obstructive SAS (central SAS or no SAS). Signs of extrathoracic upper airway obstruction (FEF50/FIF50 greater than 1) were present in 67 percent of the patients with obstructive SAS and in 71 percent of the patients without obstructive SAS. These results suggest that upper airway abnormalities, as reflected by abnormal flow volume curves, are not always associated with obstructive SAS; they favor the hypothesis of a central component in the mechanism of upper airway occlusion during sleep.

Female↗
PubMed1984

[Training respiratory muscles in subjects with chronic obstructive bronchopneumopathy].

Ten subjects with chronic bronchitis with relatively severe bronchial obstruction and, in most cases, severe distension underwent strengthening of their respiratory muscles at home by an additional respiratory resistance load for 10 minutes 3 times a day for one month. All of these subjects obtained an increased endurance of the respiratory muscles without any concomitant increase in the inspiratory muscular force. For the group as a whole, the diameter of the orifice of maximal tolerated inspiratory resistance decreased from 3.15 to 2.40 mm (p less than 0.001) and the time of endurance increased from 7 to 15 minutes 34 seconds (p less than 0.001) while the maximal inspiratory muscular pressure remained virtually unchanged. This benefit was not associated with an increase in the VO2 max SL measured during maximal effort nor with any modification of the VO2, the VE or the heart rate during sub-maximal effort; the objective benefit therefore appears to be very limited.

Aged↗
PubMed1984

[Idiopathic progressive pleuropulmonary fibrosis. Apropos of 2 cases].

The authors report two cases of retractile pleuroparenchymatous disease of the upper lobes of non known aetiology. They compare their results with those reported earlier by Stradling (20), Scadding (18), Davies (10) and attempt to identify the specific characteristics of the disease: progressive weight, loss frequent haemoptysis, apical onset, most often unilateral, with pleural involvement and progressive spread of the lesions which nevertheless remain predominantly in the upper lobes and are asymmetrical. The similarity with pulmonary fibrosis which occurs in ankylosing spondylitis is remarkable.

Aged↗
PubMed1984

[Pulmonary fibrosis and ankylosing spondylitis].

The authors report two new cases of pulmonary fibrosis associated with ankylosing spondylitis. The clinical, radiological and histological characteristics of this fibrosis are described. Various pathogenic hypotheses which could explain the onset of this fibrosis are envisaged.

Aged↗
PubMed1983

[Cockett's syndrome. Clinical and radiologic aspects].

Cockett's syndrome is due to compression of the left common iliac vein by the left iliac artery. Anatomical factors and venous anomalies contribute to the venous obstruction. Clinical symptoms only develop in cases with marked compression; they consist of functional disorders predominant in the left lower limb and, frequently, recurrent oedema. The compression may be revealed by complications such as deep thrombosis of the iliac vein or veins of the calf, or pulmonary embolism. The diagnosis is made by phlebo-ilio-cavography.

Adult↗
PubMed1982

[Mac Leod syndrome].

The Mac Leod syndrome is a unilateral pulmonary hypoplasia with emphysema. Onset usually occurs in childhood. The authors delineate this well-defined nosologic entity which is one of the many etiologies of the unilateral hyperlucent lung.

Angiography↗
PubMed1981

[Regional pulmonary function in unilateral sero-fibrinous pleural effusions and changes under the influence of treatment (author's transl)].

The regional distribution of ventilation (Vr), perfusion (Qr) (expressed in absolute values) and the regional distribution of ventilated lung volume, of ventilation and of perfusion (expressed as %) were studied using Xenon 133 in 13 subjects with a unilateral pleural effusion of proven or presumed tuberculous origin. The examinations were performed in a sitting position. The initial examination was carried out after as complete an evacuation as possible of the fluid effusion. Eight subjects underwent a follow-up examination after two months treatment with tuberculostatic agents combined with daily respiratory physiotherapy. The initial results emphasized the following points: - in absolute values, a deficit in ventilation on the involved side affecting the 3 regions explored without any similar change in perfusion ; a hyperventilation on the controlateral upper and middle regions ; - in relative values, a slight deficit in ventilated volume and perfusion localised at the base, whilst the deficit in ventilation was more marked and more extensive. After treatment the contribution to ventilation of the involved side increased on average by 6%. However this improvement only concerned the lower territory. The mechanisms of impairment of regional function and its improvement under the influence of treatment are discussed.

Adolescent↗
PubMed1977

["Immuno-allergic" complications of rifampicin therapy].

From 8 cases of immuno-allergic accidents attributed to Rifampicine, the authors review the literature on the subject. The "flue like" syndrome is the most frequent and characteristic of those accidents, worsened by haematological and renal involvements. These accidents probably belong to a pathology of immuno-complexes in relaiton with the production of anti-Rifampicine antibodies. Among the different means of in vitro diagnosis, the test with the anti-complement antiglobulin is the most reliable, though not always in good correlation with clinical signs. Though rare and most often benign, these accidents should lead to prudence in treating recurrent tuberculosis or reusing Rifampicine after a momentary interruption.

Aged↗