Biomedical subjects
Publications and source records attributed to A Poblano.
The goal of this paper is to show the paralelism between the objectives of both, neurosciences and philosophy. First, a historical review of the common works on both fields of study is made; next, the implications of the brain-mind relationship and how the use of a cerebral function theory that help us to solve the problem are discussed. Finally, the social implications of cooperation between neurosciences and philosophy are analyzed.
Thirty preschool children who survived from a neonatal intensive care unit were studied with pure tone audiometry between 125 to 8000 Hertz. Examinations were performed in a cross-sectional study at 36 to 72 postnatal months of age. Hypoacusis was found in three patients. Risk factors most frequently found in hypoacusic children were hyperbilirubinemia, hypoxia neonatorum and ototoxic exposure. All hypoacusic children had a history of preterm birth, one suffered hypoxia neonatorum, and two hyperbilirubinemia. The patients' group had an average of 2.26 risk factors. These data suggest that perinatal auditory damage occurs in the presence of additional hearing damage risk factors leading to hypoacusis.
Brainstem auditory evoked responses (BAER) were studied in Long-Evans rats in relation to development, stimulation frequency and electrode placement. Recordings made on day 11 of life showed no BAER. From day 14 on, four BAER waves were observed and centripetal maturational changes in wave latency were evident. Wave I attained adult latency values at day 20-22, wave II at day 24-25, wave II at day 24-25, and wave IV at day 39 of life. BAER latencies elicited by 8,000-Hertz tone-pips were shorter than those obtained with clicks and 4,000 Hz. Only wave II latency measures were affected significantly by electrode location.
Organic solvents affect the membrane lipids of neurons and glial cell. This led us to search for auditory damage, by means of brainstem auditory evoked responses, in subjects with chronic inhalation of thinner. Waves III and V showed delayed latency at an intensity of 90 dB, and wave V was delayed at 70 and 30 dB; the interwave intervals I-III and I-V at 90 dB and the interpeak I-V at 70 dB were also retarded. Three subjects did not show a response to stimulation at 30 dB: pure tone audiometry showed biaural mild hypoacusia in them. These results show that chronic inhalation of thinner may alter the auditory pathway of addicts.