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Biomedical subjects

A I Lukoshiavichiute

Publications and source records attributed to A I Lukoshiavichiute.

14 recordsLinked to original sources
PubMed1991

[Comparative evaluation of the effects of ethacizine and Bonnecor in the treatment of supraventricular paroxysmal tachycardia].

The efficacy of ethacizine was investigated in 46 patients, whereas that of Bonnecor++ was examined in 31 patients who suffered from frequent episodes of supraventricular paroxysmal tachycardia. After intravenous administration of ethacizine and ++Bonnecor, tachycardia was sustained in 41 (89.1%) and 14 (45.0%) cases, respectively. Their arresting effect was observed in all 17 (100%) cases of nodal tachycardia and in 19 (82.6%) cases of orthodromic tachycardia. Moreover, the effect was obtained in 3 (75.0%) cases of antidromic tachycardia and in one case of atrial ectopic tachycardia. ++Bonnecor abolished an episode of nodal tachycardia only in 3 (23.0%) patients and that of orthodromic tachycardia in 9 (56.3%). Its effect was also seen in one case of antidromic tachycardia and in one case of atrial ectopic tachycardia. ++Anti-recurrence++ effects of ethacizine and ++Bonnecor in repeated episode provocation were seen in 26 (70.5%) and 10 (32.0%) cases, respectively. Side effects occurred more frequently when ethacizine was used.

Adolescent↗
PubMed1989

[Anti-arrhythmia effectiveness of phosphobion and finoptin in the treatment of atrioventricular retrograde paroxysmal tachycardia].

Forty-four cases of induced atrioventricular retrograde paroxysmal tachycardia (AVRPT) were studied. AVRPT was provoked by means of frequent esophageal stimulation of the left atrium. During the study, records from the external and esophageal ECG leads were taken. Administration of 10 mg finoptin controlled the AVRPT attack in 86.7% of cases, and the administration of 10 mg fosfobion, in 76.2%. Antegrade block of echo impulse conduction through the atrioventricular node was the electrophysiologic mechanism of AVRPT control in all cases. The time needed to control the attack by the above-mentioned drugs ranged from a few seconds to 5 minutes. Changes of atrioventricular conductivity and patients' subjective sensations were more marked where fosfobion was used.

Adenosine Triphosphate↗
PubMed1989

[Comparative effectiveness of finoptin and phosphobion in treating attacks of supraventricular tachycardia].

Forty-seven cases of supraventricular reciprocal paroxysmal tachycardia in patients with a complementary atrioventricular junction, with retrograde flow only, were studied. An attack of tachycardia was induced by frequent transesophageal stimulation of the left atrium. Records from the esophageal lead were taken in addition to external ECG leads. An attack of supraventricular paroxysmal tachycardia could be controlled in 22 (88.0%) cases following a 10 mg intravenous dose of finoptin, and in 15 (68.2%) cases following a 10 mg phosphobion dose. The electrophysiological mechanism of the control of tachycardia by finoptin was the antegrade block of impulse conduction through the atrioventricular node in 21 (95.5%) patients, and retrograde block of the complementary atrioventricular pathway in 1 (4.5%) patient. Under the effect of phosphobion, antegrade atrioventricular block was seen in all cases. The time needed to arrest an attack by the drugs examined ranged from a few seconds to 5 minutes. The use of phosphobion was associated with more marked changes of atrioventricular conductivity and more pronounced subjective sensations.

Adenosine Triphosphate↗
PubMed1989

[Effectiveness and electrophysiologic action of amiodarone in controlling attacks of supraventricular paroxysmal tachycardia].

The study was undertaken to examine 21 patients with episodes of recurrent ventricular paroxysmal tachycardia. The latter were arrested by amiodarone intravenously injected in a dose of 5 mg/kg. The episode was stopped in 9 (45%) cases, i.e. in 6 (60%) cases with atrioventricular junction tachycardia and 3 (30%), involving accessory conduction tracts. Amiodarone-induced deterioration was observed in the atrioventricular conduction (in the anterograde one in most cases) and that along the accessory conduction tracts. In all the cases, the episode was abolished due to the anterograde block of impulse conduction along the atrioventricular node. There was an amiodarone-induced small decrease in the function of automatism in the sinus node.

Action Potentials↗
PubMed1988

[Clinical effectiveness of ethacizine in the treatment of supraventricular paroxysmal tachycardia].

I. v. administration of ethacizine was used in 2 cases of spontaneous and 15 cases of induced SV arrhythmic paroxysm. Induction was based on rapid left atrial transesophageal pacing. In addition to external leads esophageal leads were also recorded. The drug was injected at a dose of 50-100 mg. The effect was achieved in 18 (94.7%) and lasted 3-10 min. AV recurrent tachycardia was stopped as a result of blockade in the AV node in anterograde or retrograde direction, in orthodromic tachycardia--as a result of impulse conduction blockade via accessory conductive tract, and in cases of antidromic--as a result of retrograde blockade in the AV node. Among the most serious side-effects was ventricular tachycardia.

Adolescent↗
PubMed1988

[Diagnostic value of the bipolar esophageal electrocardiographic lead for detection of disorders of cardiac rhythm and conductivity at rest, during physical exercise and with Valsalva's maneuver].

Diagnosis of some heart rhythm disorders is based on bipolar esophageal ECG lead that can detect a P wave of greater amplitude, as compared to the QRS complex. The diagnostic value of bipolar esophageal ECG lead is in that it provides a more reliable assessment of pacemaker localization (both nomotopic and heterotopic), the sinoatrial node rhythm, extrasystole localization, type and mechanism of paroxysmal tachycardias, as well as antegrade and retrograde (ventricle-to-atrium) impulse conduction. Bipolar esophageal ECG lead records contribute to correct assessment of cardiac rhythm and conductivity disorders at rest as well as during exercise and Valsalva's test.

Adolescent↗
PubMed1986

[Provocation of the attack of supraventricular reciprocal paroxysmal tachycardia by frequent stimulation of the left atrium through the esophagus to determine its approximate range of reciprocal excitement].

The study involved 24 patients with supraventricular paroxysmal reciprocal tachycardia (SPRT) showing no ECG evidence of ventricular pre-excitation between the attacks. SPRT attacks could be provoked by frequent esophageal left-atrial stimulation in all patients, particularly with impulse frequencies in the 160 to 200 imp/min range, associated with first-degree atrioventricular block featuring Wenckebach's phenomenon. In most cases, induced SPRT was similar to a spontaneous attack. During an induced attack, the duration of Q--P interval was less than 70 ms in 15 (62.5%) suggesting that the scope of reciprocal excitation was limited to the atrioventricular node, and more than 70 ms in 9 (37.5%), indicating the involvement of complementary ventriculo-atrial pathways.

Adolescent↗
PubMed1986

[Recording the bundle of His potential using an esophageal electrode].

A noninvasive method for recording His bundle potential is reported that includes amplification of ECG in bipolar esophagosternal leads. A total of 106 patients were examined, with the His potential recorded in 78 (73.6%) of those. The method was validated by simultaneously recorded intracardiac electrograms in 6 patients and by atrial pacing in 3 patients. The advantages and limitations of this method are discussed.

Adult↗
PubMed1984

[Efficacy of frequent electrostimulation of the left atrium via the esophagus to stop atrial flutter].

Trans-esophagus high-frequency electrostimulation (240-520 impulses/min) of the left atrium was performed in 36 patients in order to arrest the atrial flutter. Immediately after the stimulation, the sinus rhythm was restored in 15 patients (37.5%), and 16 patients (40%) developed atrial fibrillation, 5 patients (12.5%) developed atrial flutter with irregular ventricular rhythm, in 4 patients (10%) the stimulation was ineffective. Subsequently, atrial fibrillation in 3 cases and atrial flutter in 1 case assumed the sinus rhythm. On the whole, the sinus rhythm recovered in 19 (47.5%) cases. Impulses of 20 V proved to be the most effective. This method is effective in cases of an unsuccessful drug antiarrhythmic treatment and electroimpulse therapy. There are no contraindications to this method. It can be successfully employed in cases where electroimpulse therapy is contraindicated or dangerous.

Adult↗