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Biomedical subjects

A Fujiki

Publications and source records attributed to A Fujiki.

64 records · Page 4Linked to original sources

Intermittent Wolff-Parkinson-White syndrome due to phase 3 and phase 4 block: disappearance of rapid ventricular response during atrial fibrillation.

A 65-year-old man with Wolff-Parkinson-White syndrome type A had a rapid ventricular response over an accessory pathway during atrial fibrillation. Four months later, electrophysiological study revealed the following properties: 1) The antegrade accessory pathway conduction showed phase 3 and phase 4 block. 2) The retrograde concealed accessory pathway conduction eliminated phase 4 block in the antegrade accessory pathway conduction. 3) Premature ventricular excitation arose from the accessory pathway or from the ventricular muscle close to its distal end. 4) Atrial fibrillation during isoproterenol infusion did not show rapid ventricular response. Spontaneous impairment of accessory pathway conduction due to phase 3 and phase 4 block was suspected.

Aged↗

Fast-slow type of atrioventricular nodal reentrant tachycardia: horizontal dissociation of the AV node during tachycardia.

Two patients with recurrent supraventricular tachycardia are presented. The tachycardia was initiated and terminated by atrial extrastimulation beyond the atrial relative refractory period and the atrial activation sequence during the tachycardia was low to high. The induction of tachycardia was dependent on a critical AH interval. In patient 1 who had ventriculoatrial conduction, the tachycardia was initiated by the premature ventricular stimulation followed by double atrial response. In patient 2 the ventriculoatrial conduction was not observed. In both patients, the unchanged atrial cycle length during the tachycardia with antegrade Wenckebach AH block was observed. When AH block occurred during tachycardia the first AH interval was shorter than the subsequent HA interval. In patient 2 verapamil (5 mg) prolonged the atrial cycle length during tachycardia and rapid intravenous injection of adenosine triphosphate (10 mg) terminated the tachycardia. Oral diltiazem (180 mg/day) suppressed the tachycardia in patient 1. These findings suggest that the mechanism of tachycardia may be fast-slow type of AV nodal reentry in the upper portion of the AV node and this type of arrhythmia has tendency to show incessant form.

Adenosine Triphosphate↗

Sleep of depressed patients with hypersomnia: a 24-h polygraphic study.

Sleep of 6 depressed patients with hypersomnia was studied during their depressed phase and their remitted phase using 24-h polygraphic recording. Nine normal subjects were studied as the controls. The latency to sleep onset of the depressed patients was significantly shorter than that of the remitted patients and that of the control subjects. The total sleep time of the depressed patients was significantly longer than that of the remitted patients as well as that of the controls. This increase in sleep time of the depressed patients was mainly due to the increased sleep in day time. The intervals between sleep onset and start of each sleep stage, the relative percentage of individual sleep stages, REM latency and REM density of the depressed patients were not significantly different from those of the remitted patients.

Adult↗