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Biomedical subjects

A Finsterbush

Publications and source records attributed to A Finsterbush.

At least 37 records · Page 2Linked to original sources
PubMed1982

The hypermobility syndrome. Musculoskeletal complaints in 100 consecutive cases of generalized joint hypermobility.

The hypermobility syndrome, also known as congenital laxity of ligaments and joints, occurs as an isolated condition. The genetic background is distinct from that found in other connective tissues disorders. Although joint hypermobility is relatively common in the general population, reports of musculoskeletal complaints are infrequent. An analysis of 100 consecutive cases with various musculoskeletal complaints associated with joint hypermobility demonstrated differing patterns of presentation. One group had mild musculoskeletal complaints of long duration, while another had acute episodes of pain at various sites. A third group had combined features of the first two groups. Many of the patients had complaints referable to feet and calves, while a number related to problems due to patellar hypermobility. The treatment is exercise to develop protective reflex reactions and to strengthen the surrounding musculature. Surgery must be advocated with care and is reserved for patients with secondary arthrosis.

Adolescent↗
PubMed1982

Rotational knee strain resulting in patellar dislocation. An experimental study in rabbits.

The right lower extremities of 64 young rabbits were immobilized by a plaster spica. The animals developed a gait pattern, which included internal tibial rotation and adduction of the left (unimmobilized) tibia. Twenty-one of the animals developed medial patellar dislocation in the unimmobilized lower extremity. The mechanism of the patellar dislocation in this experimental model was possibly overstretching of the lateral colateral ligament and the lateral side of the joint capsule, associated with medial rotation of the tibia and the tibial tubercle. The direction of patellar pull when gliding inferiorly during knee flexion was shifted medially, resulting in patellar dislocation and secondarily, in formation of an exostosis under the displaced patella. Hip arthrodesis in humans, as a course of rotational instability of the contralateral knee, resembles some aspects of this experimental condition.

Animals↗
PubMed1981

The cleavage intercondylar fracture of the femur.

The mechanism of injury and long-term effects in three cases suggest that a specific type of intercondylar injury of the femur, i.e., cleavage fracture, is caused by force transmitted through the patella. Treatment is conservative. The fracture does not appear to have been mentioned previously in the English literature.

Adult↗
PubMed1980

Pelvic obliquity as a factor in dysplastic hip in infancy.

Screening of the newborn to detect hip dysplasia has led to the increasing recognition of fixed pelvic obliquity, in which one hip has restriction of abduction and the other, restriction of adduction. The hip with limited abduction is apparently short, so that the infant presents signs clinically indistinguishable from those of congenital dislocation. During the years 1974-78, 192 consecutive cases of unilateral dysplastic hips were treated by us; 44 (23%) were diagnosed as having pelvic obliquity. Nine were males and 35, females; 39 were firstborn. A characteristic postural pattern was found on examination. If untreated in the early stages, a morbid degree of subluxation developed and in six cases, complete dislocation occurred. In 33 cases, complete resolution of abduction contracture and subsequent pelvic obliquity occurred within the first year of life. In a number of cases, pelvic obliquity persisted for years and in a few, resulted in physical incapacity.

Biomechanical Phenomena↗
PubMed1979

Infantile cortical hyperostosis with unusual clinical manifestations.

Infantile cortical hyperostosis may mimic a variety of serious conditions in early infancy, such as trauma, vitamin deficiencies, infection, or tumors, In a patient in good general condition there are typical roentgenological features often with involvement of the mandible that are diagnostic of the condition. In 3 cases of infantile cortical hyperostosis (Caffey's disease), one had the clinical picture of Erb's palsy and subsequently developed hyperostosis of the scapula. The second, had a "pulled elbow" and radial nerve palsy and later developed hyperostotic changes in the humerus and radius on the same side. The third was referred for treatment of congenital torticollis and found to develop hyperostosis of the clavicle on the same side. The recognition of this condition obviates unnecessary prolonged investigation and treatment.

Clavicle↗
PubMed1975

Reversibility of joint changes produced by immobilization in rabbits.

Rabbits knee joints which had been immobilized for only 2 weeks regained motion during the first 2 months of remobilization. Histologically, joint tissues were normal. Only 9 of the 14 rabbits immobilized for 6 weeks regained normal or partial knee motion. Animals that regained knee motion generally did so during the first 4 months of remobilization. There are 2 types of repair of the damaged cartilage; replacement by fibrocartilage and proliferation of chondrocytes. Normal chondrocyte alignment and normal collagen fibril orientation was not restored. Repaired cartilage did not withstand normal joint stress.

Animals↗
PubMed1975

Infantile cortical hyperostosis. Follow-up of 29 cases.

Twenty-nine cases of infantile cortical hyperostosis with a wide range of bone involvement are reviewed. Soft tissue painful swellings began before roentgenographic changes appeared in the underlying bone. In 17 babies gradual involvement of different bones was noted. In seven cases lesions previously healed became reactivated. The disease is self-limiting and appears in a narrow age group. Mandibular involvement is most common. The clinical picture and laboratory data are reported. Differential diagnosis and possible etiological factors are discussed. The similarity of infantile cortical hyperostosis to extreme periosteal new bone formation in response to trauma in sensorily deprived children is suggested as a possible etiological factor.

Bone Regeneration↗
PubMed1975

The effect of sensory denervation on rabbits' knee joints. A light and electron microscopic study.

Unilateral sensory denervation of the hind limb in a group of rabbits caused progressive atrophy of cells in all structures of the knee joint, whether or not the joint was protected by a plaster cast. Immobilization without denervation caused proliferative changes, first in the synovium and then in the articular cartilage. The initial changes in the articular cartilage following sensory denervation occurred in the middle layers, suggesting that nutritional deficiency was involved. In contrast, the first changes in the cartilage of intact immobilized limbs occurred in the superficial layers and were most likely of mechanical origin.

Animals↗