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Biomedical subjects

A Bennett

Publications and source records attributed to A Bennett.

At least 289 records · Page 16Linked to original sources
PubMed1972

Prostaglandin-like activity in ocular inflammation.

Aqueous humour samples from untreated patients with acute anterior uveitis were found to contain substantial amounts of prostaglandin-like activity. Little activity was found in aqueous from patients treated with steroids, and none (<2 ng/ml) was detected in aqueous from the uninflamed eyes of patients with cataract. Alkaline hydrolysis of the samples from inflamed eyes suggested the presence of both E and F prostaglandin-like activity. These results show that prostaglandins may be involved in acute anterior uveitis.

Alkalies↗
PubMed1972

Conditioning of a free operant in Octupus cyaneus Gray.

Operant conditioning was studied in six specimens of Octopus cyaneus Gray. An "arm-out-of-water" operant, in which the octopus inserted an arm up a feeding-tube breaking the water surface, proved susceptible to reinforcement schedules. An apparatus was developed that provided automated reinforcement and recording. Performance was studied under continuous reinforcement, fixed-ratio and variable-ratio schedules, and extinction conditions.

Journal Article↗
PubMed1971

Studies on prostaglandin antagonists.

1. Three prostaglandin antagonists have been examined for their ability to block PGE(2) and PGF(2alpha) on human, guinea-pig and isolated rat gastrointestinal muscle.2. 7-oxa-13-Prostynoic acid was either a non-selective antagonist, or was ineffective on the tissues studied; it had marked spasmogenic activity on the rat fundus.3. 1-Acetyl-2-(8-chloro-10,11-dihydrodibenz (b,f)(1,4) oxazepine-10-carbonyl) hydrazine selectively antagonized the excitatory effects of PGE(2) and PGF(2alpha) in guinea-pig and rat tissues, but not in human muscle.4. Polyphloretin phosphate selectively antagonized the excitatory effects of prostaglandins in both human and guinea-pig muscle preparations, but it caused stimulation of the rat fundus.5. All the antagonists lowered the tone in many tissues. They also reduced contractions caused by potassium.6. None of the compounds blocked the inhibitory effect of PGE(2) on intestinal circular muscle.7. The implication of these results on the nature of prostaglandin receptors, and the value of each compound as a prostaglandin antagonist are discussed.

Acetates↗