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Biomedical subjects

A Arboix

Publications and source records attributed to A Arboix.

At least 91 records · Page 5Linked to original sources
PubMed1993

[Pure motor hemiparesis secondary to chronic subdural hematoma with total recovery through medical treatment].

We discuss in this paper the cases of two patients with pure motor hemiparesis which were secondary to chronic subdural hematoma who evolved satisfactorily only with medical treatment. Of 1,000 consecutive patients with cerebro-vascular pathology admitted in our Neurology Department in the last 5 years, said two patients were the only ones (0.2%) in whom the lacunar syndrome "pure motor hemiparesis" was secondary to a chronic subdural hematoma. Such cases show that a subdural hematoma can manifest itself as a lacunar syndrome and that in selected cases the medical treatment of chronic subdural hematoma can yield goods results.

Aged↗
PubMed1992

[Cerebral venous thrombosis and hereditary protein C deficiency].

Protein C together with its plasmatic cofactor protein S and antithrombin III probably represent the most important plasmatic inhibitor in coagulation. Protein C deficiency constitutes a high risk factor for venous thrombosis. Cerebral venous thrombosis is a manifestation which is scarcely referred to in protein C deficiency. The case of a 32 year old patient with protein C deficiency is presented. The patient was admitted for an endocraneal hypertension syndrome. CT and MR demonstrated multiple hemorrhagic cerebral infarctions. Arteriography confirmed vertebral venous thrombosis. Only six cases sufficiently documenting cerebral venous thrombosis due to protein C deficiency were found in the literature. In most cases coadjuvant factors exist predisposing thromboembolic disease. The present clinical case demonstrates the importance of considering protein C deficiency in the diagnosis of cerebral venous thrombosis in young adults.

Adult↗
PubMed1991

[Lipomatous hypertrophy of the interauricular septum as a cause of cerebral embolism in a patient with isolated Wernicke's aphasia].

The clinical observation of a 74 year old male presenting isolated Wernicke's aphasia due to a temporal left infarction of cardiac embolic origin secondary to paroxysmal auricular flutter is described. Transthoracic and transesophageal echocardiography, computerized tomography and magnetic resonance provided characteristic images which permitted the establishment of the diagnosis of lipomatous hypertrophy of the interauricular septum without requiring the use of invasive techniques. Lipomatous hypertrophy of the interauricular septum is a cardiac tumor of unusual presentation constituted of an abnormal accumulation of non-encapsulated fatty tissue which occupies a thickness greater than 15 mm of the interauricular septum. Wernicke's aphasia as a form of presentation of the lipomatous hypertrophy of the interauricular septum has not previously been described in the literature.

Aged↗
PubMed1991

[Brain-heart interaction: a controlled prospective study of the electrocardiographic disorders in 100 consecutive patients with acute ischemic cerebrovascular pathology].

We carried out a prospective clinical study in 100 patients with cerebral infarctions (Cl) (56 lacunar and 44 nonlacunar infarctions) and in a control group (n = 100) without organic cerebrovascular disease to evaluate the electrocardiographic (ECG) abnormalities. 72% of the Cl and 38% of the control group had ECG abnormalities (p less than 0.0001). These abnormalities were significantly more common (p less than 0.001) at the beginning of the disease (72%) than three weeks after the development of focal neurological symptoms (54%). The major ECG findings were abnormal ventricular repolarization (changes in the ST segment and the T wave), prolonged Q-Tc interval, and U waves. Atrial fibrillation was significantly more common (p less than 0.03) in nonlacunar infarctions (18%) than in lacunar infarctions (2%) and in the control group (5%). Regarding the topography of the lesions, abnormalities of ECG were found in 80% of hemispheric infarctions, in 66% of the infarcts of basal ganglia, and in 73% of brainstem infarctions. In Cl, ECG abnormalities are common. They may be reversible, and some of them are usually correlated with the type of Cl and with the involved brain topography.

Acute Disease↗